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This breakdown in the clearance of oxidative stress leads directly to the core pathophysiology of conditions like ME/CFS and Long COVID: profound mitochondrial dysfunction

FIG 41 A and B Increased frequencies of mini-excitatory postsynaptic currents (mEPSCs) in dissociated hippocampal neurons treated with Nle

Same biological axis, opposite mechanism positions AOD-9604 is downstream with isolated effects, tesamorelin is upstream with full axis activation