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Exposure to cigarette smoke induces multiple pathological effects in the endothelium, several of which are the result of oxidative stress initiated by reactive oxygen species, reactive nitrogen species, and other oxidant constituents of cigarette smoke

We lastly wanted to determine if the smoke induced changes in endocytosis were relevant in the context of a respiratory pathogen that requires endocytosis to enter the cell using IAV as a prototypic respiratory virus

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