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To decipher the functional interactions between Keap1Nrf2 system and other oncogenic pathways is one of the most important assignments for the conquest of Nrf2-dependent cancers

The intensity of GCLC in CD8 + Ki67 + was also higher than that in CD8 + Ki67 in the IDC and MCB tumors as well as normal breast tissues (Figure 7J)

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518 Furthermore, increasing the level of available NAD + by supplementing diet containing NAD + precursors or inhibiting the activity of NAD + -dependent enzyme (e.g., PARP that snatches NAD + competing with mitochondria) is a feasible strategy to avoid the neurotoxicity related to mitochondrial dysfunction