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Besides metal homeostasis, plants posses a well-equipped antioxidative defense system to manage the metal-imposed oxidative challenge [1214]

In GPx1-deficient human endothelial cells, we found that NFB mediated the inflammatory activation at baseline, although there is a role for NFB and MAPK pathways (c-Jun N-terminal kinase or JNK and ERK1/2) in the augmented responses of these cells to TNF as well [6]

In acute AD lesions, Th17-derived cytokines such as IL-17 and IL-22 are markedly elevated (Simon et al., 2014), which aggravates skin inflammation and disrupts epidermal barrier integrity (Korn et al., 2009)