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Concluding, whatever the myotendinous junction lesions current therapy (i.e., biological scaffolds, administration of active compounds, electrospinning, self-reorganized constructs (for review see, i.e., [98])), there is still the unresolved problem of the myotendinous junction and bone-tendon junction as extremely specialized tissues, inadequate repair through mechanically and histologically suboptimal scar tissue, decreased functional properties, and greater risk of recurrent injury [98]

Several studies have found that people with high levels of homocysteine in their blood are at an increased risk of heart attack, stroke, and other cardiovascular events