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GIP (Glucose-Dependent Insulinotropic Polypeptide) Receptor Agonism: Working in concert with GLP-1, GIP activation further augments insulin release and contributes to feelings of satiety

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Moreover, the insulinotropic potency of GIP is markedly reduced in non-diabetic, first-degree relatives of T2D patients (54), suggesting altered GIP signaling could be one of the many predisposing factors for T2D later in life